(1)
Department of Emergency Medicine, Erasmus Medical Center, Rotterdam, The Netherlands
If a child has otorrhea from the myringotomy tubes, what should you do? | Topical antibiotic eardrops have high efficacy, although unusual organisms are more common for kids with tubes than for those without |
How can you remember which test is the Rinne , and which is the Weber ? | WEBER has two “Es” so it’s between the ears (the tuning fork is held at the vertex of the forehead – should hear it equally) |
How is the Rinne test performed? | Tuning fork on the mastoid, then beside the ear, in the air – Air should be heard better (still heard after the mastoid is silent) |
“Sudden” hearing loss is defined as loss of hearing that occurs over 3 days or less. What are four conduction problems that can cause sudden hearing loss? | 1. Cerumen impaction (most common) 2. Foreign body 3. TM or ossicle problems 4. Middle ear fluid |
When performing a hearing exam, how does bilateral sensorineural hearing loss present? | Bilaterally decreased hearing with normal Weber & Rinne ` |
Which medical conditions predispose the patient to sudden sensorineural hearing loss? (4) | 1. DM 2. Hyperlipidemia 3. Vascular hypercoagulable states 4. Meniere |
What is a typical environmental cause of sudden sensorineural hearing loss? | Noise |
What is a likely infectious cause of sudden sensorineural hearing loss (general category)? | Viruses (especially mumps, in unimmunized kids) |
What is a likely cause of sudden sensorineural hearing loss in a hospitalized patient? | Medication |
Do tumors cause sudden sensorineural hearing loss? | Yes – Especially if there is a small associated hemorrhage |
Which medications are most notorious for causing sensorineural hearing loss? (5 categories) | 1. Loop diuretics (especially ethacrynic acid) 2. NSAIDs 3. Salicylates 4. Certain antibiotics (e.g., gentamicin) 5. Chemo regimens |
It’s sad if you lose your hearing. How can the mnemonic “SAD” help you remember the drugs most likely to cause this problem? | SAD CHEMicals Salicylates (& NSAIDs) Antibiotics (& alcohol) Diuretics (loop) CHEMicals (reminds you of chemo regimens) |
When a patient complains of headache or ear pain, what source of the pain should always be considered? | Tooth pain |
Why is perichondritis a worrisome infection? | The infection rapidly damages the underlying cartilage – Cosmetic result is bad |
Where is perichondritis most often seen? | Pinna of the ear |
What unusual infectious agents must you watch for perichondritis? (2) | Pseudomonas & Proteus |
Which bacterium is most often identified in otitis externa? | Pseudomonas (60 %) |
Is a TM perforation an ENT emergency? | No – Follow-up with ENT later that week |
What percentage of TM perforations heals spontaneously? | 90 % |
What are the most typical or widely cited causes for TM perforation? (4) | 1. Noise 2. Barotrauma 3. Blunt or penetrating trauma 4. Lightning strike (especially if the patient is found undressed or in arrest) |
What is the hallmark of otitis externa on exam? | Pain with movement of the pinna |
What is a feared complication of otitis externa? | Malignant otitis externa |
Which patients are likely to develop malignant otitis externa? | Diabetics – 90 % of patients are diabetic (other immunodeficient patients are also at increased risk) |
What is the “triad” of Meniere disease ? | 1. Vertigo 2. Tinnitus 3. Sensorineural hearing loss (to reduce recurrences low-salt diet & hydrochlorothiazide may be helpful) |
What other patient group presents similarly to Meniere patients? | CPA tumor (cerebellopontine angle) |
What is the natural history of Meniere disease? | Intermittent recurring attacks that last weeks to years (treatment doesn’t work well, but is improving) |
Most treatments for Meniere disease focus on what aspect of the auditory system? | Reducing pressure in the endolymphatic portion of the affected ear |
What differentiates labyrinthitis from vestibular neuronitis ? | Labyrinthitis includes hearing loss! (not just vertigo or tinnitus) |
What is the most common cause of peripheral vertigo? | Benign positional vertigo (BPV) |
What are the typical features of BPV, in terms of the patients’ movement or position? | • Worse in certain positions • Worse with head motion |
What is the typical onset for BPV? | Gradual |
What is the natural course of BPV? | Spontaneous resolution |
What “key” should you find on physical exam, if you are able to elicit the vertigo of BPV? | Fatiguing (horizontal) nystagmus (fatiguing means that it decreases, then stops, on its own) |
What are the most concerning complications of sinusitis? (4) | 1. Cavernous sinus thrombosis 2. Pott’s puffy tumor (skull osteomyelitis on the forehead) 3. Orbital cellulitis 4. Brain abscess |
Sinusitis has the same typical bacterial pathogens as which other ENT infection? What are the pathogens? | • Otitis media • Strep pneumo • H. flu (non-typeable) • M. catarrhalis • Anaerobes (especially with chronic infection) ( S. pyogenes is also a common cause of otitis media, but not common in sinusitis) |
What is “ring sign” supposed to tell you? | Whether fluid dripping from the nose is snot or CSF (a ring should form around a droplet on filter paper if it’s CSF – but it’s very unreliable in reality) |
Why is a septal hematoma (in the nose) a big deal? | Because without rapid treatment the pressure causes septal necrosis – “saddle nose” deformity results |
Where do most nosebleeds come from? (give two names for it) | Anterior veins of the nose (along the septum) Or Kiesselbach’s plexus (same thing) |
Patients with posterior epistaxis make up what percentage of epistaxis patients overall? | 5 % (fortunately) |
What is the biggest risk factor for posterior epistaxis? | Arteriosclerosis |
What are the main risks involved in posterior epistaxis? (2) | 1. Hypovolemia 2. Aspiration |
How is posterior epistaxis treated? | Posterior nasal pack |
What must you watch out for with patients who have a posterior nasal pack? (4) | 1. Hypoxia & CO 2 retention (due to airway obstruction) 2. Bradycardia (vagal response) 3. Sinusitis/OM 4. Coronary ischemia (due to stress and hypovolemia, in a patient at risk for ischemia) |
What is the correct disposition for a patient with posterior epistaxis who has had a posterior pack placed? | Admit to ICU for observation under ENT’s supervision |
In cavernous sinus thrombosis , which cranial nerves are likely to be affected? | Ipsilateral 3, 4, 5, & 6 – CN6 is usually the FIRST affected, because it is not well anchored compared to the other two, so it is most easily stretched by the increasing pressure |
Which infections are likely to produce cavernous sinus thrombosis? | Midface infections – Sinusitis, periorbital cellulitis, dental |
Who was LeFort ? | A guy who dropped cadavers from heights to find out how their faces would fracture |
How did LeFort classify facial fractures? | Three groups: LeFort 1 – the maxilla moves freely LeFort 2 – the maxilla & nose move freely LeFort 3 – the maxilla, nose, & cheeks (to the orbits) move freely (in other words, the whole midface is mobile as a unit) |
Why is a LeFort facial fracture concerning? (3) | 1. Risk of airway compromise (teeth or bleeding in airway) 2. Risk of basilar skull fracture or associated c-spine injury 3. Risk of brain injury 4. Risk of tooth malocclusion if not properly repaired |
What is the most common complication of outpatient ENT surgery? | Post-op hemorrhage |
Historically, what was the most common cause of epiglottitis? | H. flu |
Which vessel is the most common culprit in posterior epistaxis? | The lateral nasal branch of the sphenopalatine artery |
How does chronic otitis media spread to other locations? | It erodes nearby bone |
What is the most common cause of sialadenitis worldwide? | Mumps |
Excruciating stabbing or electric shock-type pain to the cheek with sudden onset, that waxes & wanes, typically in a female patient = | Tic Douloureux (trigeminal neuralgia) |
If there is a hematoma on the pinna, how should it be treated and why? | • It must be aspirated (evacuated) then dressed with a pressure dressing to prevent it from refilling • Without treatment the cartilage deforms and causes cauliflower ear |
A hard, rounded swelling of the hard palate or posterior mandible that is not tender is likely to be what diagnosis? | Torus palatinus/torus mandibularis |
What is trench mouth , and what organism causes it? | • Acute necrotizing ulcerative gingivitis • Treponema vincentii Mnemonic: Think of Vincent van Gogh with bad teeth to remember the organism |
How is trench mouth treated? | Metronidazole & Penicillin (surgical debridement may also be needed) |
What is the typical age group for croup? | 6 months to 6 years (typically <3 years) |
What is the other name for croup ? | Laryngotracheobronchitis |
What infection produces “ lumpy jaw syndrome ?” | Actinomycosis (the one with “sulphur-colored crystals”) (Remember that a single lump on the jaw of an African child is usually Burkitt’s lymphoma) |
What are the most important risk factors for rhinocerebral mucormycosis ? | Neutropenia & Diabetic ketoacidosis |
A child presents with ear pain and fluid-filled blisters on the tympanic membrane. What is the most likely diagnosis and its associated organism? | • Bullous myringitis • Mycoplasma is most associated in the literature BUT the typical otitis media pathogens are actually more common |
What diagnosis and related organism should always be considered in a child who seems to have bullous myringitis? | • Ramsay-Hunt • Herpes |
The main treatment for rhinocerebral mucormycosis is . . .? | Surgical debridement (+ antifungals IV) High mortality! |
Where do preauricular sinus tracts come from? | Failure of the first & second branchial arches to fuse properly |
Why must nasal packing be removed promptly (24–48 h) after placement? | Toxic shock syndrome can develop! (The antibiotics prescribed to prevent sinusitis while the packing is in are somewhat preventative) |
Which laryngeal ring is essential in airway patency? | The cricoid (goes the whole way around) |
Which sinuses are present at birth? | Sphenoid Ethmoid (one or two cells) Maxillary (sources differ on the ethmoid – some say it is present, others dispute that) |
What is the diagnostic test of choice for neck masses? | FNA (fine-needle aspiration) |
Does anticoagulant therapy improve outcome in patients with cavernous sinus thrombosis? | No |
What study is preferred to diagnose cavernous sinus thrombosis ? | CT or MRI |
What is the most common organism found in retropharyngeal abscesses? | β-Hemolytic strep |
At what age does retropharyngeal abscess typically occur? | 6 months to 3 years |
How does retropharyngeal abscess present? | Fever Ill to toxic appearing Stridor Dysphagia +/− Drooling Refusal to eat Little movement (it hurts) |
What is the most feared complication of lateral pharyngeal space infections? | Septic thrombophlebitis of the jugular vein (Lemierre’s syndrome) |
What is the usual bacterial agent in Lemierre’s syndrome ? | Fusobacterium (others are possible, and is often polymicrobial) |
A teenager presents with a sore throat, but seems genuinely ill, with fever & rigors. What serious disorder should you consider? | Lemierre’s syndrome |
What is the most common congenital laryngeal disorder? | Laryngomalacia |
If a mandibular tumor has a “soap-bubble” appearance on X-ray, what is it? | Ameloblastoma |
What three signs should you look for on physical exam when evaluating for basilar skull fracture? | 1. Blood behind the TM (hemotympanum) 2. Raccoon eyes 3. Battle’s sign (bruising over the mastoid) |
What two presenting complaints are most common with acoustic neuromas? | 1. Hearing loss 2. Tinnitus |
What is the most common laryngeal tumor of children? | Laryngeal papillomas |
Which major artery runs through the cavernous sinus? | The internal carotid |
Adolescent male + nose bleed + nasal obstruction = | Juvenile nasopharyngeal angiofibroma |
What is the most characteristic finding on physical exam of a patient with malignant otitis externa? | Granulation tissue in the external auditory meatus |
Diplopia after facial trauma suggests what diagnosis? | Orbital floor fracture |
A patient presents with fever, malaise, and a dark red raised lesion – painful to touch – on his face. The lesion is expanding over time. What is the likely diagnosis? | Erysipelas |
Only one muscle abducts the vocal cords. Which one? | Posterior cricoarytenoid |
Infection and edema spreading from the lower part of the oral cavity into the neck is called . . .? | Ludwig’s angina – Neck is usually described as having “brawny edema” |
What usually gets Ludwig’s angina started? | Dental work |
Technically, what is Ludwig’s angina, and why is it called “angina?” | • Bilateral submandibular cellulitis • “Angina” just means “pain” (not specific to the heart) |
What types of organisms are usually involved in Ludwig’s angina? | Mixed aerobic & anaerobic |
If an item mentions “brawny edema” of the anterior neck, what diagnosis should you be thinking of? | Ludwig’s angina (Brawny just refers to the skin color deepening due to underlying infection) |
What is the most common cause of death in Ludwig’s angina? | Airway obstruction |
Can a dermoid cyst be found in the mouth? | Yes – Along the floor of the mouth |
What is the diagnostic test of choice for acoustic neuromas? | MRI with gadolinium contrast |
In general, how can you differentiate viral sialadenitis from bacterial sialadenitis? < div class='tao-gold-member'>
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